" remains a significant problem in our community today. The condition has been treated mainly with ablative procedures such as stripping and/or sclerotherapy. The aim of this study was to assess external valvular stenting (EVS) of incompetent venous valves as a reparative alternative to the management of patients with varicose veins. In addition, ultrasound examination of the superficial venous valves prior to surgery was also assessed for its ability to predict success with EVS. Methods: Valves considered for EVS were assessed with brightness-mode (B-mode), spectral pulsed Doppler (PD), col"

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"ma, and occurs in 20% to 30% of cases. The first presenting signs are sudden fever and shivering. The clinical feature is inflammatory plaque, which is often chronic and accompanied by fever. Inflammatory plaque is promoted by lymph stasis, and is marked by inflammatory episodes that often regress spontaneously. Erysipelas per se is mainly treated with antibiotics, and adjuvant therapies are not justified. The prevention of recurrence is primary. Since lymphedema is the first risk factor for recurrence, its treatment and risk of occurrence must be considered. This includes physiotherapy, well-"

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"ity, symptoms typically are a mix of cognitive and physical disturbances. Leg swelling and discomfort are one such physical symptom. The goal of the study is to define the clinical entity of a late luteal phase vasodilation syndrome in symptomatic patients. Methods: Duplex venous scans were performed in the standing position on 12 premenopausal women (age range 19-46 years) who described premenstrual symptoms of bilateral leg swelling, pressure, or pain. One scan was performed during the follicular phase (days 3-6) and one during the luteal phase (days 20-24). Great saphenous vein (GSV) dia"

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"e of the world’s top authorities in clinical research on venous thromboembolism, and he has published several landmark articles in this field. In his current article he discusses the ways in which recurrences of venous thromboembolism can be prevented. This is a problem of very high practical interest for doctors, as recurrent episodes of venous thrombosis are among the deciding risk factors in the development of postthrombotic syndrome. Seshadri RAJU, from Jackson, Mississippi, is one of the great pioneers in the treatment of proximal outflow obstructions by venous stenting. He discusses"

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"Raju - Phlebolymphology - 2008; 15 (1): 12 Pelviperineal venous insufficiency and varicose veins of the lower limbs V. Stvrtinova - Phlebolymphology - 2008; 15 (1): 17 Skin necrosis as a complication of compression in the treatment of venous disease and in prevention of venous thromboembolism S. Raju - Phlebolymphology - 2008; 15 (1): 27 LYMPHOLOGY Towards a better understanding of lymph circulation O. Stücker, C. Pons-Himbert, E. Laemmel - Phlebolymphology - 2008; 15 (1): 31 SERVIER FELLOWSHIP The Second American Venous Forum, Servier Traveling Fellowship R. W. Quan - P"

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"years of follow-up. This risk is higher in patients with permanent risk factors for thrombosis, such as active cancer, prolonged immobilization because of disease, and antiphospholipid antibody syndrome; in patients with idiopathic presentation; and in carriers of several thrombophilic abnormalities, including carriers of AT, protein C or S, increased factor VIII, hyperhomocysteinemia, homozygous carriers of factor V Leiden or prothrombin G20210A variant, and carriers of multiple abnormalities. Patients with permanent risk factors for thrombosis should receive indefinite anticoagulation, consi"

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"become clear that the lesion is present in over half the general population in silent form. A clinical syndrome variously known as May- Thurner syndrome, Cockett syndrome, or “iliac vein compression syndrome”, caused by NIVL, is thought to be a rare form of chronic venous disease (CVD). However, with liberal use of intravascular ultrasound (IVUS), the lesion is found in over 90% of highly symptomatic CVD cases, with a very broad clinical and demographic spectrum. Silent NIVL in the general population may play a permissive role in the development of CVD. Venous stenting of NIVL provides exc"

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"ficiency (PVI) in women to date has primarily involved its most familiar clinical presentation, ie, pelvic congestion syndrome (PCS) accurately described by Hobbs.1 For the last few years,2 more attention has focused on involvement of PVI in the pathogenesis of primary varicose veins or recurring varicosities of the lower limbs. This finding has been confirmed by a national epidemiologic survey that evaluated the potential incidence of pelvic pain of venous origin in a targeted population of women, and its possible association with lower limb varicosities.3 While advances in the recognition"

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"kin allergic reaction and eczema,1 and nerve palsy,2 have been reported, but the most severe is skin necrosis in diabetics or patients with peripheral arterial disease of the lower limbs. There is a consensus for contraindicating compression in patients whose ankle/brachial index (ABI) is less than 0.6, but the arterial disease is not always identified. Diabetes also carries a potential risk of skin wound, but few data are available. LITERATURE REVIEW A literature search for skin necrosis related to compression identified several articles,3-6 including the Scottish survey.7 The aim of this"

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"which appeared much more crucial. In the last decade, researchers have become interested in lymphatic function since many diseases seem to interact with it (cancer, inflammation, infection, auto-immunity). The lymphatic system is harder to study than its vascular counterpart as its vessels are ill-defined, almost invisible. Intravital microscopy alone correctly visualizes these structures, thus shedding light on their function and quantifying their movements. This paper focuses on lymph anatomy and physiology, summarizes research trends, and considers lymph diseases and the latest treatments, "

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